Publications

Included here is a list of publications from OCG programs. All published data are available to the research community through the program-specific data matrices.

* denotes publications from the CTD2 initiative that are results of intra-Network collaborations

 

CTD²
August 20, 2014
Clinical Cancer Research

The authors used a functional kinomics approach to identify novel survival kinases involved in G2-M cell-cycle checkpoint, SFK, PI3K, and FAK pathways in head and neck squamous cell carcinoma.

CTD²
August 15, 2014
Genes and Development

By screening a custom library of shRNAs directed toward known drug targets in a genetically defined Myc-driven HCC model, researchers identified cyclin-dependent kinase 9 (Cdk9) as required for disease maintenance of hepatocellular carcinoma.

CTD²
August 11, 2014
Cancer Cell

A review of a recent finding connecting the genes GF1 and GF1B in medulloblastoma.

CTD²
August 09, 2014
Nature Protocols

Researchers develop protocol to construct genetic interaction map through two-stage approach.

CTD²
August 07, 2014
Cell

The authors performed an integrative analysis on 3,527 specimens from 12 cancer types, revealing a unified classification into 11 major subtypes.

CTD²
July 30, 2014
Neuro-Oncology

In this review researchers describe basic and translational research into glioma proliferation and invasion, in particular the phenotypic switch underlying a stochastic "go or grow" model of tumor cell behavior.

CTD²
July 28, 2014
The Journal of Experimental Medicine

Researchers discuss the emerging themes of CAF biology in the context of tumorigenesis and therapy.

CTD²
July 24, 2014
Cancer Research

The results suggest RAC1 P29S status may offer a predictive biomarker for RAF inhibitor resistance in melanoma patients.

CTD²
July 18, 2014
Cell Reports

The findings presented in this manuscript establish CTCF as a prominent tumor-suppressor gene and point to CTCF-mediated epigenetic stability as a major barrier to neoplastic progression.

CTD²
July 17, 2014
Cell

Researchers elucidate endogenous barriers limiting the process of reprogramming somatic cells to induced pluripotent stem cells by systematically dissecting human cellular reprogramming.

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